Free radic biol med

Elsevier Inc. How to publish in this journal. The set of journals have been ranked according to their SJR and divided into four equal groups, four quartiles, free radic biol med. Q1 green comprises the quarter of the journals with the highest values, Q2 yellow the second highest values, Q3 orange the third highest values and Q4 red the lowest values.

Free Radical Biology and Medicine is a peer-reviewed scientific journal and official journal of the Society for Redox Biology and Medicine. The journal covers research on redox biology, signaling, biological chemistry and medical implications of free radicals , reactive species, oxidants and antioxidants. This article about a biochemistry journal is a stub. You can help Wikipedia by expanding it. See tips for writing articles about academic journals. Further suggestions might be found on the article's talk page. Contents move to sidebar hide.

Free radic biol med

.

It is based on the idea that 'all citations are not created equal'. You can help Wikipedia by expanding it. The purpose is to have a forum in which general doubts about the processes of publication in the journal, experiences and other issues derived from the publication of papers are resolved, free radic biol med.

.

The discovery of vitamin E will have its th anniversary in , but we still have more questions than answers regarding the biological functions and the essentiality of vitamin E for human health. Discovered as a factor essential for rat fertility and soon after characterized for its properties of fat-soluble antioxidant, vitamin E was identified to have signaling and gene regulation effects in the s. In the same years the cytochrome P dependent metabolism of vitamin E was characterized and a first series of studies on short-chain carboxyethyl metabolites in the s paved the way to the hypothesis of a biological role for this metabolism alternative to vitamin E catabolism. Recent findings are consistent with gene regulation and homeostatic roles of these metabolites in different experimental models, such as inflammatory, neuronal and hepatic cells, and in vivo in animal models of acute inflammation. Molecular mechanisms underlying these responses are under investigation in several laboratories and side-glances to research on other fat soluble vitamins may help to move faster in this direction. Other emerging aspects presented in this review paper include novel insights on the mechanisms of reduction of the cardiovascular risk, immunomodulation and antiallergic effects, neuroprotection properties in models of glutamate excitotoxicity and spino-cerebellar damage, hepatoprotection and prevention of liver toxicity by different causes and even therapeutic applications in non-alcoholic steatohepatitis. We here discuss these topics with the aim of stimulating the interest of the scientific community and further research activities that may help to celebrate this anniversary of vitamin E with an in-depth knowledge of its action as vitamin. Abstract The discovery of vitamin E will have its th anniversary in , but we still have more questions than answers regarding the biological functions and the essentiality of vitamin E for human health.

Free radic biol med

Cardiovascular diseases CVD , including ischemic heart diseases and cerebrovascular diseases, are the leading causes of morbidity and mortality worldwide. Atherosclerosis is the major underlying factor for most CVD. It is well-established that oxidative stress and inflammation are two major mechanisms leading to atherosclerosis. Under oxidative stress, polyunsaturated fatty acids PUFA -containing phospholipids and cholesterol esters in cellular membrane and lipoproteins can be readily oxidized through a free radical-induced lipid peroxidation LPO process to form a complex mixture of oxidation products. Overwhelming evidence demonstrates that these oxidized lipids are actively involved in the inflammatory responses in atherosclerosis by interacting with immune cells such as macrophages and endothelial cells. In addition to lipid lowering in the prevention and treatment of atherosclerotic CVD, targeting chronic inflammation has been entering the medical realm. Clinical trials are under way to lower the lipoprotein a Lp a and its associated oxidized phospholipids, which will provide clinical evidence that targeting inflammation caused by oxidized lipids is a viable approach for CVD.

Draculaura monster high

Download as PDF Printable version. Journal homepage Online access. Scope Free Radical Biology and Medicine is the premier forum for publishing groundbreaking research in the redox biology of both health and disease. Toggle limited content width. Cites Year Value External Cites per document 4. Please help improve this article by adding citations to reliable sources. Contents move to sidebar hide. The set of journals have been ranked according to their SJR and divided into four equal groups, four quartiles. Year Documents International Collaboration accounts for the articles that have been produced by researchers from several countries. Further suggestions might be found on the article's talk page.

Federal government websites often end in. The site is secure.

Article Talk. Kelvin Davies. Join the conversation about this journal. Journal Self-citation is defined as the number of citation from a journal citing article to articles published by the same journal. Hidden categories: Articles lacking sources from October All articles lacking sources Articles with short description Short description is different from Wikidata Biweekly journals infobox Articles with outdated impact factors from Pages using infobox journal with unknown parameters All stub articles. All types of documents are considered, including citable and non citable documents. Please help improve this article by adding citations to reliable sources. Publisher Elsevier Inc. Elsevier Netherlands. Toggle limited content width. Elsevier Inc. Categories : Biochemistry journals Biweekly journals English-language journals Elsevier academic journals Biochemistry journal stubs. Read Edit View history. Cites Year Value External Cites per document 4.